logo
School of Biomedical Sciences
   
   
  

Molecular signalling

Importance of Research in Molecular Signalling

Just as humans populate the world in which we live, inside each cell there is a world populated with proteins.  Like us, proteins are born, they have specific jobs to do and ultimately they die and get replaced. Proteins are also social, they work in interactive groups; they are efficient at what they do and part of the reason is that they communicate with each other, directly and indirectly.

 

Our Research Interests in Molecular Signalling

The ability of proteins to interact and communicate is the basis of molecular signalling. It is going on all the time in and between all the cells of our bodies and we take it completely for granted – until something goes wrong. Cancer is a good example of molecular signalling gone wrong, but equally, aberrant molecular signalling underlies the development of hereditary and degenerative diseases, such as Paget’s disease of bone, rheumatoid arthritis and Parkinson’s disease. Our work is aimed at understanding the fundamentals of molecular signalling so we, and others, can develop strategies to correct things when they go wrong.

For more information, please email Professor Peter Shaw .

See research degree opportunities related to this group.

 

Group Publications

Chromosome 'by-Aurora-ientation' during mitosis

Description
Cell Biol Int; Wheatley, S.P.

PU.1 is a major transcriptional activator of the tumour suppressor gene LIMD1

Description
FEBS Lett; Foxler, D.E., James, V., Shelton, S.J., Vallim, T.Q., Shaw, P.E., and Sharp, T.V.

Impact of p62/SQSTM1 UBA domain mutations linked to Paget's disease of bone on ubiquitin recognition

Description
Biochemistry; Garner, T.P., Long, J., Layfield, R., and Searle, M.S.

Genomic DNA damage and ATR-Chk1 signaling determine oncolytic adenoviral efficacy in human ovarian cancer cells

Description
J Clin Invest; Connell, C.M., Shibata, A., Tookman, L.A., Archibald, K.M., Flak, M.B., Pirlo, K.J., Lockley, M., Wheatley, S.P., and McNeish, I.A.

A point mutation in the ubiquitin-associated domain of SQSMT1 is sufficient to cause a Paget's disease-like disorder in mice

Description
Hum Mol Genet; Daroszewska, A., van 't Hof, R.J., Rojas, J.A., Layfield, R., Landao-Basonga, E., Rose, L., Rose, K., and Ralston, S.H.
 
 

School of Biomedical Sciences

University of Nottingham
Medical School
Queen's Medical Centre
Nottingham, NG7 2UH

telephone: +44 (0) 115 823 0141
email: enquiries